Joseph Bisson
jab2069@med.cornell.edu
Postdoctoral Fellow
My overall research goal is to uncover novel mechanisms of cardiogenesis to gain a greater understanding of congenital heart disease. In particular, I’m interested in how homozygous (and heterozygous) GATA4, GATA5, and GATA6 loss-of-function mutations impact cardiac development using both zebrafish and human embryonic stem cell model systems.
jab2069@med.cornell.edu
Postdoctoral Fellow
My overall research goal is to uncover novel mechanisms of cardiogenesis to gain a greater understanding of congenital heart disease. In particular, I’m interested in how homozygous (and heterozygous) GATA4, GATA5, and GATA6 loss-of-function mutations impact cardiac development using both zebrafish and human embryonic stem cell model systems.
Education
|
PhD
BS |
University of Rochester
University of New Hampshire |
2017
2011 |
Publications
|
2015
|
Ajima R., Bisson J.A., Helt J.C., Nakaya M., Habas R., Tessarollo L., He X., Morrisey E.E., Yamaguchi T.P., Cohen E.D. Disheveled associated affector of morphogenesis 1 and 2 are co-required for myocardial maturation and sarcomere assembly. Dev Biol. 2015; 408(1):126-139.
Bisson J.A., Mills B., Paul Helt J.C., Zwaka T.P., Cohen E.D. Wnt5a and Wnt11 inhibit the canonical Wnt pathway and promote cardiac progenitor development via the Caspase-dependent degradation of AKT. Dev Biol. 2015;398(1):80-96. |